Final draft of NIH cell phone exposure study in rats does not show any brain tumor risk.

The final draft (not yet the officially published version) of the "TOXICOLOGY AND CARCINOGENESIS STUDIES IN Hsd:SPRAGUE DAWLEY SD RATS EXPOSED TO WHOLE-BODY RADIO FREQUENCY RADIATION AT A FREQUENCY (900 MHz) AND MODULATIONS (GSM AND CDMA) USED BY CELL PHONES " is out. Unlike the preliminary, midstudy data, there is no brain cancer or brain tumor risk found.

The Sprague-Dawley breed of rats seems prone to cardiac tumors, and there is a hint of an increase in the rats of those cardiac tumors in male but not female rats. Since, on the whole, these cardiac tumors do not tend to have any gender bias in rats in other studies, one finds even those results suspect.

Overall, there seems to be no real reason to avoid the use of cell phones because of cancer risk. I think that the study authors are writing as if they are really searching for a positive result in a study that had little to show.




The predominant source of human exposure to radio frequency radiation (RFR) occurs through the use of cellular phone handsets. The Food and Drug Administration nominated cell phone RFR emission for toxicology and carcinogenicity testing in 1999. At that time, animal experiments were deemed crucial because meaningful human exposure data from epidemiological studies were not available. Male and female Hsd:Sprague Dawley SD rats were exposed to time-averaged whole-body specific absorption rates of Global System for Mobile Communications (GSM)- or Code Division Multiple Access (CDMA)-modulated cell phone RFR at frequencies of 900 MHz (herein referred to as “cell phone RFR”) in utero, during lactation, and after weaning for 28 days or 2 years. Genetic toxicology studies were conducted in rat peripheral blood erythrocytes and leukocytes, brain cells, and liver cells.

Under the conditions of this 2-year whole-body exposure study, there was some evidence of carcinogenic activity of GSM-modulated cell phone RFR at 900 MHz in male Hsd:Sprague Dawley SD rats based on the incidences of malignant schwannoma in the heart. The incidences of adenoma or carcinoma (combined) in the prostate gland, malignant glioma and benign or malignant granular cell tumors in the brain, adenoma of the pars distalis in the pituitary gland, pheochromocytoma (benign, malignant, or complex combined) in the adrenal medulla, and pancreatic islet cell adenoma or carcinoma (combined) may have been related to cell phone RFR exposure. There was no evidence of carcinogenic activity of GSM-modulated cell phone RFR at 900 MHz in female Hsd:Sprague Dawley SD rats administered 1.5, 3, or 6 W/kg. There was some evidence of carcinogenic activity of CDMAmodulated cell phone RFR at 900 MHz in male Hsd:Sprague Dawley SD rats based on the incidences of malignant schwannoma in the heart. The incidences of malignant glioma in the brain, adenoma of the pars distalis in the pituitary gland, and adenoma or carcinoma (combined) of the liver may have been related to cell phone RFR exposure. There was equivocal evidence of carcinogenic activity of CDMA-modulated cell phone RFR at 900 MHz in female Hsd:Sprague Dawley SD rats based on the incidences of malignant glioma in the brain and pheochromocytoma (benign, malignant, or complex combined) in the adrenal medulla.

Increases in nonneoplastic lesions in the heart, brain, and prostate gland of male rats, and of the heart, thyroid gland, and adrenal gland in female rats occurred with exposures to GSM cell phone RFR at 900 MHz. Increases in nonneoplastic lesions of the heart, brain, and prostate gland occurred in males, and of the brain in females exposed to CDMA cell phone RFR at 900 MHz.

Terror exposure and headache.

Stress has long been identified as a major causal factor in the occurrence of headaches. A recently published study in Norway looked at the survivors of a terrorist's attack on a boys camp in that country. Reference: HERE. This study showed a 4 fold increased incidence of tension and migraine headaches for those with terrorism exposure.

Charles Peirce on speculative aspects of the theistic argument from reason

It is not that such phenomena might not be capable of being accounted for, in one sense, by the action of chance with the smallest conceivable dose of a higher element; for if by God be meant the Ens necessarium, that very hypothesis requires that such should be the case. But the point is that that sort of explanation leaves a mental explanation just as needful as before. Tell me, upon sufficient authority, that all cerebration depends upon movements of neurites that strictly obey certain physical laws, and that thus all expressions of thought, both external and internal, receive a physical explanation, and I shall be ready to believe you. But if you go on to say that this explodes the theory that my neighbour and myself are governed by reason, and are thinking beings, I must frankly say that it will not give me a high opinion of your intelligence. But however that may be, in the Pure Play of Musement the idea of God's Reality will be sure sooner or later to be found an attractive fancy, which the Muser will develop in various ways. The more he ponders it, the more it will find response in every part of his mind, for its beauty, for its supplying an ideal of life, and for its thoroughly satisfactory explanation of his whole threefold environment.

--- Charles Peirce, 1908; MS 841 and P1166; CP 6.452-91; EP2:434-450

Choke faux 'choke

The recent winter storms have caused some early, juvenile green 'ulu fruit fall from the ma'afala ulu tree. Maryruth cooks and peels it to make a very artichoke-heart flavored side dish.

A new cover-up REVEALED!

Well, actually, it was an old cover-up, from the nineteen-sixties. Nevertheless, it was a cover-up! Conspiracy theory lovers, rejoice! The sugar industry seems to have covered up evidence sugar increases cancer risk. A news perspective HERE.



Sugar industry sponsorship of germ-free rodent studies linking sucrose to hyperlipidemia and cancer: An historical analysis of internal documents

Cristin E. Kearns, Dorie Apollonio, Stanton A. Glantz

PLOS Biology 15(11): e2003460

Published: November 21, 2017


In 1965, the Sugar Research Foundation (SRF) secretly funded a review in the New England Journal of Medicine that discounted evidence linking sucrose consumption to blood lipid levels and hence coronary heart disease (CHD). SRF subsequently funded animal research to evaluate sucrose’s CHD risks. The objective of this study was to examine the planning, funding, and internal evaluation of an SRF-funded research project titled “Project 259: Dietary Carbohydrate and Blood Lipids in Germ-Free Rats,” led by Dr. W.F.R. Pover at the University of Birmingham, Birmingham, United Kingdom, between 1967 and 1971. A narrative case study method was used to assess SRF Project 259 from 1967 to 1971 based on sugar industry internal documents. Project 259 found a statistically significant decrease in serum triglycerides in germ-free rats fed a high sugar diet compared to conventional rats fed a basic PRM diet (a pelleted diet containing cereal meals, soybean meals, whitefish meal, and dried yeast, fortified with a balanced vitamin supplement and trace element mixture). The results suggested to SRF that gut microbiota have a causal role in carbohydrate-induced hypertriglyceridemia. A study comparing conventional rats fed a high-sugar diet to those fed a high-starch diet suggested that sucrose consumption might be associated with elevated levels of beta-glucuronidase, an enzyme previously associated with bladder cancer in humans. SRF terminated Project 259 without publishing the results. The sugar industry did not disclose evidence of harm from animal studies that would have (1) strengthened the case that the CHD risk of sucrose is greater than starch and (2) caused sucrose to be scrutinized as a potential carcinogen. The influence of the gut microbiota in the differential effects of sucrose and starch on blood lipids, as well as the influence of carbohydrate quality on beta-glucuronidase and cancer activity, deserve further scrutiny.

Jabuticaba, vintage 2017.

Or is that an arborage?